Hey, they might be our new overlords soon.I like your "may have"?
Hey, they might be our new overlords soon.I like your "may have"?
I should take care indeed, telling truth is not good in the west, but not getting better either in the old kingdom ?Hey, they might be our new overlords soon.
Hold down on it and uninstall will pop up - at least in Android, if you have an iphone you are on your own )Now the covid alert systems aren't being used does anyone know how to remove them from an android device? I have opened app manager but the uninstall icon isn't highlighted, so it wont uninstall.
Thanks Knobby, you're a champion mate.?Hold down on it and uninstall will pop up - at least in Android, if you have an iphone you are on your own )
Omicron probably manufactured in lab
Amazed they admitted it. It was obvious after a month that it didn't stop the spread.
The recently identified, globally predominant SARS-CoV-2 Omicron variant (BA.1) is highly transmissible, even in fully vaccinated individuals, and causes attenuated disease compared with other major viral variants recognized to date1–7. The Omicron spike (S) protein, with an unusually large number of mutations, is considered the major driver of these phenotypes3,8. We generated chimeric recombinant SARS-CoV-2 encoding the S gene of Omicron in the backbone of an ancestral SARS-CoV-2 isolate and compared this virus with the naturally circulating Omicron variant. The Omicron S-bearing virus robustly escapes vaccine-induced humoral immunity, mainly due to mutations in the receptor-binding motif (RBM), yet unlike naturally occurring Omicron, efficiently replicates in cell lines and primary-like distal lung cells. In K18-hACE2 mice, while Omicron causes mild, non-fatal infection, the Omicron S-carrying virus inflicts severe disease with a mortality rate of 80%. This indicates that while the vaccine escape of Omicron is defined by mutations in S, major determinants of viral pathogenicity reside outside of S.
Why are LABS still getting away with this research? We were told there would be no more Chimeric cutting and pasting research?This work was supported by Boston University startup funds (to MS and FD), National Institutes of Health, NIAID grants R01 AI159945 (to SB and MS) and R37 AI087846 (to MUG), NIH SIG grants S10-OD026983 and SS10-OD030269 (to NAC), Peter Paul Career Development Award (to FD), and BMBF SenseCoV2 01KI20172A (AE) and DFG Fokus COVID-19, EN 423/7-1 (AE).
MickTo test the role of the S protein in Omicron phenotype, we generated a chimeric recombinant virus containing the S gene of Omicron (USA-lh01/2021) in the backbone of an ancestral SARS-CoV-2 isolate (GISAID EPI_ISL_2732373)30 (Fig. 1c). To produce this chimeric Omi-S virus, we employed a modified form of cyclic polymerase extension reaction (CPER) (Extended Data Fig. 1) that yielded highly concentrated virus stocks, containing 0.5-5 × 106 plaque-forming units (PFU) per ml, from transfected cells within two days of transfection (Fig. 1d,e), obviating the need for additional viral amplification31,32.
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